Sentence examples for mouse fibroblasts exhibited from inspiring English sources

H1 from ras-transformed mouse fibroblasts exhibited higher phosphorylation than did their untransformed counterparts [ 25].

Similarly, H-Ras transformed mouse fibroblasts exhibited low mitochondrial respiration, an increased dependency on glycolysis, a sensitivity to glycolytic inhibitors, and an insensitivity to OXPHOS inhibitors [148].

Consistent with the need for effective repair of DSBs by NHEJ, XRCC4- or LIG4-deficient mouse fibroblasts exhibit marked sensitivity to ionizing radiation, growth defects and premature senescence [ 25, 26].

Similarly, Brca2-deficient mouse embryonic fibroblasts exhibited weak SAC activity.

STAT3-knockout mouse embryonic fibroblasts exhibited prolonged IL-6-mediated STAT1 activation and induction of IFN-γ-inducible genes [ 48].

For example, deletion of SIRT3 in mouse embryonic fibroblasts exhibited the phenotype of high proliferation, antiapoptosis, and the characteristic of invasion and metastasis [ 11].

WrnΔ hel /Δ hel mutant mouse embryonic fibroblasts exhibit increased oxidative stress.

Cardiac fibroblasts exhibit high metabolic flexibility.

Wild type and TEF fibroblasts exhibit very different morphologies.

26 PGC-1α KO fibroblasts exhibit a decrease in SOD2, catalase, and GPx1 mRNA content relative to WT fibroblasts and PGC-1α KO mice were more vulnerable to oxidative stress.

This is different to previous in vitro studies carried out by our lab and others of human and mouse fibroblasts; in these studies, FRDA cells exhibited increased levels of FAST-1 expression relative to control cells (De Biase et al., 2009; Sandi et al., 2014).