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Mouse exposure to UFP.
Pursuant to the above, in the mouse, exposure to perfluorooctane sulfonic acid (PFOS) caused a variety of immunotoxic consequences, including decreased immunoglobulin response to a standard antigen challenge [ 4, 5].
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According to studies on mice, exposure to PAH causes gains in fat mass.
Similar to saline injected mice, exposure to PDGF alone had no significant effect on neuronal survival.
In male infant mice, exposure to B[alpha]P induces liver tumors [ 17].
In rats or mice, exposure to ELF-MF did not affect the serum testosterone level [ 20, 35, 38, 58].
Also in 1-month-old mice exposure to dox resulted in transgene expression in articular cartilage and growth plate.
These findings indicate that mice exposure to a sublethal dose of TBI (4.0 Gy) could cause long-term BM injury.
This suggests that in mice, exposure to the Aβ fragment cause impairments in the learning and memory systems.
In wild-type mice, exposure to orally administered docetaxel was six-fold lower compared to Abcb1a/1b knockout mice (Bardelmeijer et al, 2002).
In mice, exposure to 17α-estradiol during a critical period of reproductive tract development leads to subsequent gynecologic malignancies (Hajek et al. 1997).
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