Sentence examples for mouse experiments revealed from inspiring English sources

Gnotobiotic mouse experiments revealed that increased protein intake can limit microbial drug inactivation.

Indeed, knockout mouse experiments revealed that the absence of MMP-9, but not of MMP-2, reduces arthritis progression [ 39].

Mouse experiments revealed that the mutation of Arg61 to Thr, or of Glu255 to Ala, may reduce the domain interactions.

Both the in vitro and in vivo mouse experiments revealed that renal Klotho production is not affected by hyperglycemia per se.

Mouse coinfection experiments revealed that the bacterial load in the post-lavage lung tissue and bronchoalveolar lavage (BAL) was comparable between the coinfected (IAV+T4) and single T4 infected groups until 6 hours post infection (hpi) (Fig. 2a and b).

The mouse model experiments revealed that SIRT1 overexpression significantly enhanced metastatic nodules.

Subchronic T3 treatment also enhanced the number of β-galactosidase immunopositive cells within the neocortex of Shh+/LacZ reporter mice, and colocalization experiments revealed that the expression of β-galactosidase was neuronal.

The ChIP PCR product comprised of 109 bp starting from +1581 of the 2891 bp mouse VEGFA intron1.The ChIP experiments revealed that following addition of thapsigargin, ATF4 was recruited to the VEGFA intron 1 (Figure 5I).

Arginine butyrate-treated mice gene expression profiling experiments revealed that several genes that control cell proliferation, growth and differentiation are differentially expressed consistent with its histone deacetylase inhibitory activity when compared to control (saline-treated) mdx mice.

Bromo-deoxyuridine (BrdU) incorporation experiments revealed that mouse NS cell cultures contained a higher percentage of BrdU labelled cells at all checked time points, reaching a plateau close to 100% after ∼3 days incubation (Figure 5B and 5C).

Furthermore, use of gene-deficient mice and adoptive cell transfer experiments revealed that ILC2s were necessary and sufficient to mount NMU-elicited type 2 cytokine responses.