Sentence examples for mouse erythroid cells from inspiring English sources

Whyatt D, Lindeboom F, Karis A, Ferreira R, Milot E, Hendriks R, et al. An intrinsic but cell-nonautonomous defect in GATA-1-overexpressing mouse erythroid cells.

Correlation between gene silencing and localisation to transcriptionally repressive heterochromatic compartments has been reported in mouse cycling lymphocytes [55], [56], [57], human and mouse erythroid cells [58], [59], [60] and retinoblastoma cells [61].

Furthermore, we observe that the boundaries of the topological domain, which contains the β-globin locus, are easily observed in mouse erythroid cells.

A molecular colony technique recently developed by Gavrilov et al. [ 17] investigated multicomponent interactions among remote enhancers and active β-globin genes in mouse erythroid cells.

In silico analysis revealed that the HRE sequences within the KCC3b and KCC4 promoters are conserved in mice and humans; thus, we examined whether PlGF regulated the expression of KCC3 and KCC4 mRNA in mouse erythroid cells in vivo.

In mouse erythroid cells, the active α1 and α2 genes are in close spatial proximity of the flanking RHBDF1, MPG and c16orf35, including the cis-regulatory MREs, as the result of erythroid-specific changes in the chromatin conformation [ 59].

Considering the number of human ES cell lines that have been established so far, the intensive testing of a number of these lines for erythroid potential may allow the establishment of human erythroid cell lines similar to the mouse erythroid cell lines described here.

We further applied dCaP to detect differential TAL1 occupancy in response to GATA1 restoration in two mouse erythroid cell lines.

We further show in the mouse dataset that dCaP captures genomic regions showing significant signal variations for TAL1 occupancy between two mouse erythroid cell lines.

Mouse ENOCDE Data: we obtained read counts by the ChIP-seq experiments of TAL1 in two mouse erythroid cell lines (G1E, G1E-ER4) from Wu et al. [ 15, 30].

The target ChIP data and the supporting data are generated from the same mouse erythroid cell line with restoration of GATA1 function (G1E-ER4).