Sentence examples for mouse embryos whereas from inspiring English sources

It is generally accepted that paternally imprinted X inactivation occurs exclusively in extraembryonic lineages of mouse embryos, whereas cells of the embryo proper, derived from the inner cell mass (ICM), undergo only random X inactivation.

Ssea-1 is expressed in the ICM of mouse embryos, whereas it is not expressed in human embryos [ 31].

Zscan4d is expressed predominantly in the two-cell stage mouse embryos, whereas Zscan4c is expressed predominantly in the mouse embryonic stem (ES) cells.

KRIT1−/− (K−/−) and KRIT1+/+ (K+/+) mouse embryonic fibroblast (MEF) cell lines were established from KRIT1−/− and KRIT1+/+ E8.5 mouse embryos, respectively, whereas KRIT1 9/6 (K9/6) MEFs were obtained by infecting KRIT1−/− cells with a lentiviral vector encoding KRIT1 (Lv-KRIT1) [12].

Recent genetic evidence demonstrates that Cdk1 is the only Cdk sufficient to drive the mammalian cell cycle because embryos from Cdk1-/ mice fail to develop to the morula and blastocyst stages, whereas mouse embryos lacking all interphase Cdks (Cdk2, Cdk3, Cdk4 and Cdk6) undergo organogenesis and develop to midgestation [ 33].

Whereas early mouse embryos can tolerate a lack of dosage compensation, X inactivation becomes essential soon after implantation [ 39].

Brg1 −/− mouse embryos die at the preimplantation stage, whereas Brm −/− mice develop normally [ 17].

KEGG pathway analysis of all of the networks involved with these genes revealed that the regulation process in pig embryos is mostly associated metabolic pathways whereas that in mouse embryos, is mostly associated with signal transduction (Additional file 2: Figure S10C and Additional file 9: Table S8).

In addition, genetically engineered mice conditionally overexpressing VEGF-C showed hyperplasia of lymphatic vessels, whereas VEGF-C-null mouse embryos completely lacked lymphatic vasculature [ 15].

In the development of the lymphatic system, the role of VEGF-D is dispensable [ 139], whereas VEGF-C null mouse embryos completely lack a lymphatic vasculature and die prenatally [ 140].

An example is the universal ribosomal protein L38, whose mutations cause unique patterning defects in mouse embryos, including homeotic transformations of the axial skeleton, whereas mutants of several other ribosomal proteins show no such defects [29,31].