Sentence examples for mouse embryos revealed from inspiring English sources

Painstaking cell transplantation experiments in these early mouse embryos revealed that canonical Wnt signals and Dkk1 function as repulsive and attractive guidance cues, respectively, in the migration of visceral endoderm cells.

Whole mount in situ hybridization of embryonic day (E) 9.5 and 10.5 mouse embryos revealed strong positive signals for snx3 mRNA in the forebrain, pharyngeal arches, eyes, and limb buds.

For example, an allelic series of mutations in Lrp5 and 6 in mouse embryos revealed that the Lrp5 loss in combination with Lrp6 loss produces a more severe phenotype than Lrp6 loss alone, and that Lrp6 loss is more severe than Lrp5 loss alone [22], [45].

Analysis of Sall2-deficient mouse embryos revealed delayed apposition of the optic fissure margins and the persistence of an anterior retinal coloboma phenotype after birth.

In situ hybridization of mouse embryos revealed MIA/CD-RAP expression starting from the beginning of chondrogenesis and abundant throughout cartilage development.

Examination of the mouse embryos revealed the development of spina bifida occulta, an incomplete closure and deformation of the vertebrae [69].

Fadloun, A. et al. Chromatin signatures and retrotransposon profiling in mouse embryos reveal regulation of LINE-1 by RNA.

Specifically, analyses in mid-gestation mouse embryos reveal a marked elevation of H4K20me3 in muscle and neural lineages and a corresponding decrease in H4K20me1 as cells became post-mitotic [21].

Summary: Single-cell RNA-sequencing of human and mouse embryos reveals conserved and human-specific transcriptional programmes as well as a functional requirement for TGFβ signalling in human embryos.

Smoother than silk polymers, 100 year old blood vessels and the head of a mouse embryo revealed in exquisite detail.

Fate mapping studies of the mouse embryo reveal that progenitors of the definitive or gut endoderm are recruited from the epiblast during gastrulation and they are incorporated into a pre-existing population of visceral endoderm which may also contribute to the tissues of the embryonic gut.