Sentence examples for mouse embryogenesis led from inspiring English sources

Cardiac-restricted deletion of Dicer during early mouse embryogenesis led to embryonic lethality due to cardiac malformations, while deletion later in development led to an early post-natal lethality secondary to a dilated cardiomyopathy [14], [15].

While no studies have been done on the role of Shh signaling during postembryonic development in mammals, transgenic inhibition of Shh signaling in the intestine during mouse embryogenesis leads to severe defects in the villus/crypt structure around neonatal stages, indicating a critical role of Shh signaling for intestinal embryogenesis.

There is limited evidence on the role of this protein in cancer, but results of Pofut2 knockout mice showed that the loss of the protein leads to epithelial-mesenchymal transition in mouse embryogenesis, suggesting an important role of the protein in cancer [ 41].

Embryonal carcinoma (EC) cell lines are models for early cells in mouse embryogenesis.

We analyzed the formation of LVVs at the molecular and ultrastructural levels during mouse embryogenesis and identified three critical steps.

The present study was designed to identify enhancers responsible for the dynamic expression pattern of Six1 during mouse embryogenesis.

All of these adhesion molecules might play overlapping roles with CD146, and could thus be able to compensate for its deletion in vivo during mouse embryogenesis, resulting in normal physiological angiogenesis in CD146EC-KO mice.

During mouse embryogenesis GATA-4 is expressed first in primitive endoderm and then in definitive endoderm derivatives, including glandular stomach and intestine.

The protein is expressed throughout the mouse embryogenesis and is strongly upregulated in brain and developing structures of the immune system in the course of development.

Here, we report the role of TIAR during mouse embryogenesis.

Mutation of the widely expressed oto gene causes loss of the anterior forebrain during mouse embryogenesis.