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The Florida mouse displays a soft and silky pelage, brown above and orange on the cheeks, shoulders, and lower sides.
This suggestion is in accordance with the fact that the Nxnl1−/− mouse displays a progressive loss of cones [36].
Consequently, PPARγ+/− mice display a significantly higher bone mass due to increased bone generation (Akune et al., 2004).
Indeed, LAB mice display a deficit in hypothalamic AVP [25].
In addition, the mutant mice display a microphthalmia phenotype.
However, LRP5 knockout mice display a relatively normal vascular network in the ganglion cell layer.
In contrast, the LAMP-2 knockout mice display a more severe phenotype.
Accordingly, TGFβ1-deficient mice display a severe defect in LC, but not in DC, development [21].
Homozygous prestin knockout mice display a 40 60 dB hearing loss [9].
However, under environmental factors that cause stress, mutant mice display a hypoactive behavior.
AID deficient mice display a mild B cell hyperplasia and enlarged germinal centers [6].
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CEO of Professional Science Editing for Scientists @ prosciediting.com