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Computations were performed across 1,444 mouse datasets, and the genes were clustered hierarchically according to average correlation coefficients.
Highly significant correspondence was observed between the FPD, CBF and mouse datasets and the genes switched on after irradiation of lymphoblasts.
We performed the co-expression search with these 15 genes on 12 ES-related mouse datasets and recorded both individual and aggregated search results.
Previous models were based on yeast and mouse datasets and were more specific, see e.g. [ 27] that models the C16-branch of sphingolipid metabolism in RAW264.7 cells.
The meta-analysis provided 40 consensus integromics modules across mouse datasets and revealed microRNA relations with substantial collective action during aging.
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The mouse datasets is extracted from a set of combined SNPs from the 10 K GNF (http://www.gnf.org/) mouse dataset and the 140 K Broad/MIT mouse dataset (Wade and Daly, 2005).
These were then combined with the wild mouse dataset and a PCA was performed on this combined dataset.
They suggest that the inclusion of about 24%99%9% posterior probability interval 17.4 30.6%) of all cassette exons in our mouse dataset and 30%99%9% posterior probability interval 26.2 34.9%) of all cassette exons in our human dataset is dependent on the TSSs of the corresponding transcripts.
Using a human-hamster RH panel, a mouse-hamster RH panel, an aneuploid mouse dataset and publicly available TCGA data, we present strong evidence that many genes possess the ability to decrease their gene expression in response to increased copy number (i.e., possess negative cis α).
In addition to the above-mentioned mapping of the 5 mouse datasets (E18 and P7 cortices, and adult brain, liver and muscle), we also mapped the 4 human RNA-seq datasets (hESC, N1, N2 and N3) onto the mouse reference genome (mm9 in the UCSC database; [ 21]).
The two mouse datasets – MmBMM and MmRAW – share 10,113 genes.
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