Sentence examples for mouse cells have from inspiring English sources

Mouse cells have a history of not always being a good model for human cells.

Basically, the human cells taken from a blastocyst (a five- or six-day embryo) have already moved on to the next stage, where equivalent mouse cells have not.

Mouse cells have a special chromatin-binding protein that alters recombination hotspots: it binds to highly evolved genome expression signals in mouse DNA and protects them from disruption by homologous recombination.

Precursor cells from rat hippocampus have been propagated with FGF2 alone, but mouse cells have resisted this technique.

These differentiated mouse cells have been shown previously to improve the amphetamine-induced rotational behavior in unilaterally 6-OHDA-lesioned and not immunosuppressed rats after intrastriatal transplantation [3].

As the parental mouse cells have an active X chromosome it was anticipated that silencing of the human X would not be deleterious; however the induced human XIST failed to localize to the X chromosome, and thus it was not surprising that no silencing of human X-linked genes was observed.

After normalization to the level of p24, virus released from mouse cells had 6 10 fold lower infectivity than that from human T cells (Figure 8B).

A 1000-fold increase iNanogog following reprogramming in mouse cells has been observed (Theunissen et al., Current Biol 2011).

Inactivation of the OGT gene in mouse cells has shown that OGT is required for embryonic stem cell viability and mouse ontogeny (Shafi et al, 2000).

Expression profiles of Drosophila and mouse cells having reduced SIN3 levels revealed that several nuclear encoded mitochondrial genes are subject to SIN3 regulation [ 8, 9].

For example, previous work in mouse cells has suggested that loss of Prdx1 promotes the oxidative inactivation of PTEN and increases Akt activity (Cao et al., 2009).