Sentence examples for mouse brains show from inspiring English sources

Previous studies demonstrated that neural stem cells (NSCs) isolated from the subventricular zone (SVZ) of Atm-/ mouse brains show defective self-renewal and proliferation, which is accompanied by activation of chronic p38 mitogen-activated protein kinase (MAPK) and a lower level of the polycomb protein Bmi-1.

Fibroblasts from NPA patients and aSMase-knockout mouse brains show similar autophagolysosome accumulation and impaired autophagy.

HSVtk gene-transduced Muse cells (Muse-tk cells) at a cell number of only 1/32 that of U87 human glioma cells completely eradicated U87 gliomas in nude mouse brains, showing a robust in vivo bystander effect.

The biochemical analysis of PrPres in TgOvPrP4 mouse brains showed a fully conserved pattern, compared to that in the natural host, with three distinct PrPres products.

White light illumination of the explanted mouse brains showed a distorted anatomy, a loss of normal white and gray matter structures of normal mouse brain, and increased tissue volume at the site of the tumor implantation.

Expression profiling studies of HD mouse brains showed that hundreds of genes were deregulated, including a conspicuous decreased expression of neuronal specific genes in the striatum [9], [10], [11], [12].

No pretreatments aimed at eliminating PrPC were used; disease-associated PrP deposits (called PrPSc in the present study) in infected mouse brains showed a distinctly different appearance after immunostaining than PrPC, which, if at all, appeared as faint homogenous background staining.

Immunoprecipitated BACE1 from Mgat3 +/+ and Mgat3−/− mouse brains showed comparable enzymatic activity in vitro (Fig 5A).

FAT of the suckling mouse brains showed a positive reaction for lyssavirus antigen.

The postmortem analysis of the mouse brains showed that the GFP signal in rAAV-injected mice (P0) was 5-fold stronger than the signal for Venus of rAAV-SHANK2-R462X rAAV-SHANK2-R462X rAAV-SHANK2-R462Xinjected.  5A and C).

Despite the existence of TGF- β1 in all the observed fields, intense staining was detected in the dense whorls of meningioma cells, whereas normal mouse brains showed very little or no reactivity.