Sentence examples for mouse brain whereas from inspiring English sources

The authors found that the majority of macrophages are resident microglia (CX3CR1+/CCR2−) in the normal mouse brain, whereas only CCR2+/CX3CR1− macrophages, the monocyte-derived macrophages from the peripheral blood, are found in the GSC-derived GBM xenografts.

However, our findings indicated a lack of GSK3β staining in most hypothalamic areas of the adult mouse brain, whereas previously strong labeling of GSK3β was noted in this region of the brain in the adult rat [28].

The subcellular distribution and oligomeric complexes of dynamin GTPases are not altered by modulating LRRK2 in mouse brain, whereas mature OPA1 levels are reduced in G2019S PD brains.

Eighty-four miRNAs have significant expression alterations in mouse brain, whereas only 56 miRNAs have significant expression alterations in the liver.

These results indicate that the administration of D-ribose is able to trigger hyperphosphorylation of Tau in mouse brain, whereas D-glucose cannot.

Notably, as in humans, the genes from the yellow module are also enriched in the early embryonic mouse brain whereas genes from the brown module are enriched in the late embryonic mouse brain (supplementary material Fig. S5C).

The TDP-43 inclusions showed nuclear clearance and cytoplasmic accumulation in the knock-in mouse brains whereas the TDP-43 inclusions showed nuclear localization in the cortex of control knock-in mice (Figure 8E,F).

25– 27 Our observation that Ta1505 antibody suppressed not only Ta1505- but also PHF-1-staining in mouse brains whereas Ta4 and Ta9 antibodies only reduced PHF-1-staining suggests that tau phosphorylation occurs first at Ser413 and then at Ser396.

This is in line with data of our experiments, where bands corresponding to the C2 fragments appeared to decrease in Prnp+/+ ischemic mouse brains, whereas an increase of bands corresponding to the C1 fragments was visible compared with Prnp+/+ sham controls.

The rationale for these particular concentrations derives from previous in vivo studies demonstrating that a 4 mg/kg i.p. injection of METH results in a concentration of about 10 µM in the mouse brain [45], whereas a 1 mg/kg i.v. infusion also results in a similar concentration in rats [46].

The SH3 domains of CIN85, amphiphysin II and EEN-B1 were able to retain endogenous OPHN1 from adult mouse brain extracts, whereas neither GST alone nor GST-SH3 domain of βPIX, a Rac/Cdc42 exchange factor known to interact with PAK3 (23), were able to bind to OPHN1 (Fig. 1C).