Among the tissues examined in PLP mice, brain, lung and thymus showed no reduction in weights as compared to control tissues.
Figure 3 referred to the mean value of SOD inhibition activities in Tx and Rx for mice brain, lung, and liver tissues.
The results obtained from the experimental analysis of MDA levels in mice brain, lung, and liver tissues are presented in Figure 1.
In liver tissue, there was a significant difference between Tx (68.50 ± 1.82%) and Rx (59.13 ± 2.0%) with P = 0.04. Figure 4 shows the levels of GSH content in mice brain, lung, and liver tissues.
Similarly, total RNA was isolated from mouse brain, muscle, lung, spleen, heart, liver, kidney, and testis.
Interestingly, cells and tissues from p27 null mice, including brain, lung, and retina, present an elevated basal expression of Sox2, suggesting that p27 contributes to the repression of Sox2.
In response to anoxia, an increase in VEGF concentration and an altered pattern in the expression of VEGF receptor have been found in hippocampal neurons in vitro and in mouse brain, kidney, testis, lung, heart, and liver in vivo [24] [26].
For specificity, milk antibody was pre-adsorbed to an acetone extract of E16 mouse embryos and adult mouse brain, liver and lung.
In contrast, a previous study had shown that Hif-1α mRNA in rat and mouse brain, kidney and lung was up-regulated within 30 min of hypoxia treatment in vivo, but returned to baseline after 4 h (Wiener et al., 1996).
On the contrary, the results obtained on thin sections of unirradiated mice tissues including brain, lung, heart, and small intestine have shown that they are almost completely negative for γH2AX (Rübe et al. 2008).
