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These vascular networks were perfused with mouse blood indicated by perfused lectin staining in human CD-31 positive vessels.
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Similarly, the protein concentration of thrombi formed from NK1 receptor deficient mouse blood was 79.7% (± 5.5%) lower than that of thrombi formed from litter-matched control mouse blood, indicating an important role for NK1 and substantiating data on human platelets using L733060.
Platelet adhesion tests using mice blood indicated that vascular scaffolds containing 0.5% GO had low platelet adhesion and activation.
Bimatoprost was found as the intact molecule in mouse skin and blood, indicating that it stimulates hair growth by interacting with prostamide-sensitive receptors 8– 13.
The blood indicated something else.
Dengler, L. et al. Cellular changes in blood indicate severe respiratory disease during influenza infections in mice.
Moreover, mice engineered to generate smaller than normal quantities of SIRT1 carried relatively little fat in their blood, indicating that their cells hung onto it.
However, as indicated by the mouse blood DA TC and Human AZA TC data sets, a p-value of 0.01 or less does not always yield high correlations (Fig. 3a).
We also found that the transplanted tissues contained mouse CD31+ cells forming duct-like structures (Fig. 7B), which indicated that transplanted tissue also contained mouse blood vessels.
Parabiosis experiments have established that old mouse brains are improved by exposure to young mouse blood.
For mouse blood sugars we used an ACCU-CHEK Aviva blood glucose meter (Roche, Indianapolis, IN).
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