Sentence examples for mouse analyses indicating from inspiring English sources

Prh−/− ES cells do not contribute to the formation of lymphoid or myeloid tissues in chimaeric mouse analyses, indicating that PRH is required for the formation of the HSCs [ 55].

Interestingly, while both substances revealed comparable cytotoxic effects on human ESCs in this study, only ATRA but not 13CRA were shown to be cytotoxic in mouse ESCs in previous analyses, indicating the necessity of developing hESC-based assays for the assessment of human-specific developmental toxicity.

Previous analyses indicate mice are a suitable system to monitor differences in virulence if survival curves opposed to LD50 are analyzed [ 16- 18].

Although the basic cell types and laminar distribution patterns of Otx2 and PV antibody labeling in the KO mice were similar to those in the control mice (see above), the following quantitative analyses indicated significant differences between the control and KO mice. Figure 8(a) indicates distributions of average diameter of the Otx2-IR cells in the control and KO mouse SC.

Additional pathologic analyses indicated that mice in the ANXA2 shRNA group died due to tumor formed at the splenic bed (local regional tumor growth) (Figure S7).

Since WRN protein will affect the topology of the chromosomes [ 18] and SAFB1 is involved in chromatin organization [ 22, 23], we expected to see increased DNA damage in cells lacking the functions of both proteins with deleterious consequences on health span of mice, even though previous spectrometry analyses indicated that both proteins may not interact directly [ 28].

These analyses indicate that only mouse cells maintained under physiological O2 concentrations develop a SASP that is quantitatively and qualitatively similar to the human SASP.

Our biochemical, histological and molecular analyses indicate that in mice IL-6 contributes to the MCD diet-induced liver inflammation.

These analyses indicated that Rcan2−/− mice had similar energy expenditure as weight-matched controls.

Quantitative analyses indicated that 28 d mice exposed to hyperoxia during the neonatal period had 88% more macrophages in the lung than did 28 d RA controls).

Initially, suppression of STAT3 and β-catenin expression was observed in the Gan mouse model of gastric cancer, and subsequent immunohistochemical analyses indicated suppression of STAT3 phosphorylation.