In BALB-neuT664V-E IFN-γ knockout mice, absence of IFN-γ leads to accelerated progression of neu carcinogenesis [ 26].
In mice, absence of ZFP57 also results in defective establishment of maternal methylation at the imprinted Snrpn locus [3,5].
80 In essra-null mice, absence of estrogen-related receptor alpha increased modestly osteoblast differentiation and cancellous bone mineral density, as well as mesenchymal cell differentiation into osteoblasts.
In young mice, absence of λ5 expression led to a similar increase in PC reactivity among bone marrow and splenic B cells.
In mice, absence of Six1 leads to the reduction in size or loss of multiple organs as a result of decreased proliferation and increased apoptosis [ 5- 10].
Interestingly in adult mice, absence of gap junction coupling between astrocytes and oligodendrocytes, also mediated by Cx43 and Cx30, may also lead to altered potassium buffering by oligodendrocytes, and further increase [K+]o.
In adiponectin-deficient mice, absence of adiponectin was associated with a 2-fold increase in leukocyte-rolling and a 5-fold increase in leukocyte adhesion in the microcirculation [ 48].
In a previous study, we observed that in global and Purkinje-cell-specific knockout mice, absence of FMRP leads to enhanced parallel fiber long-term depression (Koekkoek et al. 2005).
Will the mouse's absence satisfy him entirely?
A single probe test was given on the tenth day, in which the platform was removed from the maze in the mouse's absence.
Four probe trials were given on the tenth day, in which the platform was removed from the maze in the mouse's absence.
