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And motor dysfunction was improved after autologous transplantation of BMSCs.
Motor dysfunction was evaluated with the UHDRS and a total functional capacity score, TFC [8].
Prior to testing (training or trial) animals were acclimatized to the testing room for 30 min. Forelimb motor dysfunction was assessed using a grip strength meter (GSM TSE Systemss, Bad Homburg, GER) as previously described [11], [33].
Because motor dysfunction was not a "conditio sine qua non" in former diagnostic criteria of CRPS, many studies do not report the amount of motor changes in CRPS or confine to describe manifestations of existing motor deficits [1], [45], [46].
The patient with motor dysfunction was withdrawn from treatment after receiving two doses of atacicept.
The onset and progression of motor dysfunction was quantified using several different tests.
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In addition to olfactory deficits, motor dysfunction is another prominent manifestation of these conditions.
However, data regarding its protective role in stroke associated motor dysfunction is scarce.
3-NP-induced striatal lesion size and motor dysfunction were significantly increased in A-CREB mice compared to controls.
The first signs of motor dysfunction are easily discernible when mice lose their typical hyperactivity, develop constant body clasps, display abnormal extension of the hindlimbs during a tail suspension test and a few days prior to death, also show pronounced weight loss, dorsal kyphosis and resting tremor18.
Whole-cell patch clamp recordings were performed on layer 2/3 primary motor cortical pyramidal cells and parvalbumin interneurons from BACHD mice at 3 months, when the mice begin to demonstrate mild motor dysfunction, and at 6 months, when the motor dysfunction is more severe.
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