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The motor dysfunction recovered gradually within 7 days, whereas balance improved more slowly.
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Endothelial dysfunction recovered after acidosis correction.
Pre-existant organ dysfunction recovered.
The nerve dysfunction recovered slowly in the year following the end of lengthening.
All animals had recovered from the TBI-associated motor dysfunction prior to performing the cognitive testing.
And motor dysfunction was improved after autologous transplantation of BMSCs.
No overt motor dysfunction or behavioral impairment was observed.
Motor dysfunction will not develop because most of the lower extremity motor nerves originate above the S2 segment of the spinal cord [8].
Complications related to motor dysfunction during lengthening or union time (fracture or bending of the regenerate bone) were recorded.
In addition to olfactory deficits, motor dysfunction is another prominent manifestation of these conditions.
huANG treatment increased survival and delayed motor dysfunction as assessed by rotarod in SOD1G93A mice.
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