Sentence examples for motor cortex damage from inspiring English sources

The capellini handling test is a simple and quantitative way to estimate paw and digit function after motor cortex damage.

Mutism has been frequently observed in cases of insular pathology: Transient mutism is found in cases of left inferior motor cortex damage extending to the insula [ 57, 58], whereas lasting mutism appears to be associated with bilateral lesions of the frontal operculum and anterior insula [ 59- 61].

38 57–59 When the motor cortex is damaged (eg, congenital brain lesion), action observation and imitation may influence cortical reorganisation by directly restoring the damaged motor pathways or reinforcing other pathways that originally helped to perform motor actions, or both.

Motor deficits associated with CCI injury are likely the result of direct damage to motor cortex; however, subcortical regions are also damaged, such as CC and caudate-putamen, which may all contribute to motor dysfunction.

A new approach utilising action observation to stimulate the mirror neuron system (MNS) offers another opportunity to stimulate the damaged motor cortex before the infant has achieved volitional reach and grasp.

However, when motor cortex on one side is damaged peri-nataly, control of the contra-lesional hand can be taken over by the intact, ipsilateral motor cortex.

The precise mechanism that links damage of the motor cortex to this complex, impulse-driven behavior is not known.

For example, increasing the concentration of FGF2 protein appears to offset the functional outcome of neonatal damage to the motor cortex (Monfils et al. 2005), and the neuroprotective effect of nicotine in animal models of Parkinson disease (Quik and Di Monte 2001) is associated with its ability to up-regulate fgf2 expression (Belluardo et al. 2004).

Both CST and motor cortex are common sites of developmental brain damage leading to cerebral palsy (Eyre 2007).

This damage typically included parts of the motor cortex, lateral dysgranular retrosplenial cortex, and parietal cortex (above dorsal CA1), often extending caudally to reach visual areas.

In other words, task-related brain activation was greater in left primary motor cortex (leg) in subjects with SCI with greater cord damage.