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If MTrPs are one of the main reasons for shoulder pain (active MTrPs) and altered motor activation patterns (active and latent MTrPs), as several authors have proposed, then anti-inflammatory treatment [ 26, 48, 70] and muscle strengthening exercises should not be the treatment of first choice.
How does the matching of motor activation patterns occur?
While the participants included in this study had no current symptoms they may still have dysfunctional motor activation patterns which have not presented symptomatically.
Experimental muscle pain, clinical muscle pain, and MTrPs have all been shown to alter motor activation patterns in a similar manner as the kinematic disturbances seen in shoulder pain patients often referred to as SIS [ 47- 49].
Lucas et al. showed that subjects who received myofascial dry needling, followed by passive muscle stretching to remove latent MTrPs, showed normalized motor activation patterns within 20 to 30 minutes following the treatment [ 48].
Therefore, it is reasonable to expect that treatment of MTrPs may lead to normalization of motor activation patterns and may facilitate spontaneous recovery of shoulder pain, either without exercising or by making exercise more effective.
Similar(54)
Both experimentally induced and spontaneous muscle pain lead to an aberrant motor activation pattern that is also present in patients with shoulder pain [ 68, 69].
Because the skeletal muscle is an elastic entity, functional movements are part of variations in the neuro-motor activation pattern.
There were no significant differences between the baseline motor-related activation patterns in the three sessions.
We first contrasted the motor-related activation patterns before and after each real tDCS condition compared with sham i.e. [ tDCSPost tDCSPre)–(shamPost shamPre)] using a voxel-wise analysis.
Given the apparent influence of CNTF genotype on muscle strength [ 4], we hypothesized that subjects with G/A genotype would demonstrate a different motor unit activation pattern with increasing muscle force generation in the vastus medialis than homozygotes with G/G genotype.
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