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Control of gastrointestinal motility is very complex involving multiple signals, such as nitric oxide (NO), gastrin, ghrelin, prostaglandins, 5-hydroxytryptamine (5-HT), dopamine, catecholamine, and acetylcholine [ 3, 4].
While the core protein regulatory machinery involved in ciliary motility is very likely to be conserved, some variations in response to increased Ca2+ between single cell ciliates and mammalian cilia have been reported [ 90].
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These slowest velocities are randomly directed and contribute negligibly to the standard in vitro motility velocity except when directed movement is very slow.
This indicated the effect of prodigiosin in bringing down the motility capacity which is very essential factor for pathogenicity.
Therefore, evaluation of esophageal motility is a very important factor in assessment of this condition; however, esophageal manometry has rarely been reported in this context [8, 9].
The distinctive features of myosin VIIa (long run lengths, slow motility) will be very useful in video-based single-molecule applications.
Sperm motility parameters are very important semen characteristics.
In this context, Snail1 induced motility has been very recently reported to be mediated by Rho GTPases [36].
Moving in the direction of an attractive site or factor is typical for embryonic movements and metastatic dissemination of cancer cells and motility strategies are very similar for both categories.
As MIF-II strongly inhibits forward motility of mature sperm and its action is very specific (Fig. S2), it has the potential to be served as a contraceptive.
This reflex leads to inhibition of the duodenal motility in response to distension of the stomach and is very likely involved in gastric emptying.
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