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"Kinesin motility is driven by subdomain dynamics". eLife 6 (1): e28948.
Neurite motility is driven by actin polymerization; however, the mechanisms are not clearly understood.
Sperm motility is driven by flagellar ATP-dependent dynein motor proteins.
Swarming motility is driven by flagellar rotation in a film of fluid on the surface of the substrate [7].
This motility is driven by CCR7-binding chemokines and may be pivotal for T cells to find their proper partners among numerous other cells.
Cell motility is driven by remodeling of the actin cytoskeleton and cell contacts with the extracellular matrix (ECM) [1], a process which is under the control of a plethora of actin-binding proteins.
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These two distinct forms of motility are driven by the same physical (i.e. flagella) and chemotactic machineries (10, 11), however, swarming is strictly preceded by a morphological differentiation of short swimmer cells into elongated, multinucleated and hyperflagellated swarmer cells.
There are indications that gliding and FMG1-B motility are driven by the same process (Bloodgood, 2009), because they move at comparable speeds and both require ligation of the major flagellar surface protein, FMG1-B, into large clusters (Bloodgood and Workman, 1984), accompanied by a Ca2+-dependent signaling pathway (Bloodgood and Salomonsky, 1994).
Twitching bacterial groups also produce traction hotspots, but with forces around 100 pN that fluctuate rapidly on timescales of <1.5 min. Gliding, the second motility mechanism, is driven by lateral transport of substrate adhesions.
The motility of amoeboid sperm is driven by the regulated assembly and disassembly of major sperm protein (MSP) cytoskeleton [ 7, 8].
The invasive process of Entamoeba histolytica, the etiologic agent of human amebiasis, is driven by motility, and cytoskeleton dynamics is indispensable for parasite invasion [ 9].
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