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Most tissue studies of the pathophysiology of BD are conducted in skin.
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Both systemic and topical administration of the dendron to tumour-bearing mice induced higher porphyrin levels than the widely investigated hexyl ester derivative in most tissues studied, although it was not possible to surpass the levels induced by 5-aminolaevulinic acid.
Porphyrins with side chains with nett cationic character also exhibited up upward (parabolic) dependence on pi for most tissues studied, with maximal porphyrin localisation at pi of 0-0.5.
These data indicate that exercise likely results in tissue-specific adaptation of the telomere maintenance pathways; however, regardless of tissue-specific effects, it is important to note that exercise results in a phenotype that is teloprotective in most tissues studied to date.
However, for most tissue engineering studies the cell population needs to be expanded.
Amplification was carefully assessed by fluorescence in situ hybridization on tissue microarrays containing triplicate 2 mm cores/sample, which is far more tissue than used in most tissue microarray studies.
Unfortunately, quantification studies have usually not been done at a functional (e.g., protein level) level for most tissue-based studies.
Most hitherto published tissue studies in dogs and cats, however, underscore even baseline neuroanatomical accuracy and lack reproducible sampling schemes.
In addition to studying two processes that occur in most tissues, we studied the kinome requirement for a muscle specific process.
Most elastic tissues studies in literature have used the strain energy function approach.
Most tissue culture and in vivo studies have instead shown that Wnt signalling is either required for, or cooperates with, other mutations to overcome senescence (Delmas et al, 2007).
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