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If the panelists look like the people who are going to be sitting across from you in upcoming negotiations, and on most panels of industry experts they will, the odds are it's going to be a big fat waste of your time.

Figure  3 and most panels of Fig.  4 show that adding extra intervals to the B trace is more effective in lowering the DLs than adding extra looks to the A trace.

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In the bottom-most panels of Figs. 5A and 5B we present the time-delay as a function of the input frequency for various magnitudes of receptor downregulation/desensitization.

To illustrate this, we show in Fig. 11 the same figure as in the right-most panel of Fig. 10, but with I ( t ) = 20 t e − t replaced by I ( t ) = 7 t e − t.

This is not surprising: In the left-most panel of Fig. 12, in the range when M Δ is an increasing function of Δ, the duration of the input spike volley, M Δ Δ, is much greater than 3, the decay time constant of the excitatory synaptic input pulses.

The right-most panel of Fig.  1 shows a slightly different situation.

I think an argument in this direction can be made based on the right-most panel of Figure 7.

The left-most panel of Figure 1 d shows the checkerboard pattern of two distinct antibodies: anti-VSV-G (high intensity Atto 740) and anti-HA (low intensity Atto 740).

doi 10.1038/sj.bjc.6603837 Owing to an error by the author in the algorithm for computing red meat intake, the values for the cut points and the hazard ratios and confidence intervals in the bottom-most panel of Table 2 were incorrect.

I think an argument in this direction can be made based on the right-most panel of Figure 7 – it would seem that the downstream (+16 through about +40) footprint changes characteristic of the complete complex still appear when Pol II is added after the inhibitor, but the TATA to +1 changes are reduced.

In Figure 5A (left most panel), histology of liver and spleen has been replaced.

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