At 1500 K, most of the defects fail to make excursions from their defect sites resulting in diffusion.
Our evaluation shows that even simple system's specifications developed by relatively experienced developers are prone to defects, and our approach is successful in uncovering most of these defects.
In this case, most of the defects near the ion incident direction were vacancies, and the defects on the other side were almost interstitials.
However, further analysis showed that most of these defects are stress-induced, presumably arising as a secondary consequence of previously identified defects in the gut.
Most of the defects concentrate at the end-of-range regions where the ions stop.
Most of the defects are oval on the right and left ventricular aspects with variable size.
Most of these defects are caused by resection of oral squamous cell carcinoma (OSCC) and the biologic effects of rhBMP-2 on these carcinoma cells are unknown.
With an appropriate adjustment of the molding parameters it was possible to eliminate most of these defects, and better reliability was achieved.
Furthermore, noting that most of these defects occur in structures of neural crest origin, we hypothesize that poorly controlled maternal diabetes interferes with cephalic neural crest cell migration.
X-ray tomography was used to explore cracks, voids, and fiber bundles distortion and to reconstruct 3D finite element (FE) meshes containing most of these defects.
