Sentence examples for most frequent resistance from inspiring English sources

In this study and in other studies, the most frequent resistance phenotype was that of streptomycin for Escherichia coli, Klebsiella pneumonia, and Proteus species [29].

H274Y (the most frequent resistance mutation in human influenza A) was seen in four isolates in N1 and one isolate in N2.

The most frequent resistance mechanism detected in S. pneumoniae is represented by associated mutations of both gyrA and parC genes [ 1, 3, 7].

The next most frequent resistance phenotypes were resistance to kanamycin, nalidixic acid, tetracycline and enrofloxacin at 11.8%, and kanamycin and ampicillin at 8.8%.

The importance of this aspect of RNA polymerase physiology is clearly illustrated by the fact that one of the most frequent resistance mutations observed in the clinic, S450L, (equivalent to S531L in Escherichia coli) also carries the lowest fitness cost [ 45].

The most frequent resistances of MRSH isolates was demonstrated to benzylpenicillin (91.7%), erythromycin (91.7%), gentamicin (75.0%), tetracycline (66.7%), fluoroquinolones (41.7%) and co-trimoxazole (41.7%).

The most frequent resistances of MRSH isolates demonstrated were to benzylpenicillin (91.7%) with the presence of the blaZ gene; erythromycin (91.7%) and clindamycin (41.7%) with the presence of ermA, ermC, and msrA genes; and gentamicin (75.0%) with the presence of aac(6′ -Ie-aph(2")-Ia and aph(3′)-IIIa genes.

In the most frequent drug resistance profile, the resistance proteins P-glycoprotein/MDR1 (MDR1, ABCB1), thymidylate-synthetase, glutathione-S-transferase-π, metallothionein, O-methylguanine-DNA-methyltransferase and major vault protein/lung resistance-related protein were up-regulated.

In the most frequent drug resistance profile (cluster 4) all resistance proteins investigated (MDR1, TYMS, GSTP1, MT, MGMT, MVP/LRP) were up-regulated.

In STEC strains, the most frequent drug-resistance combination was resistance to ampicillin, cefalothin, and tetracycline, which was detected in 8 isolates, once with resistance to amoxicillin/clavulanic acid and once with resistance to gentamicin.

The characteristics of this antitumoral activity include: High rate of apoptosis, especially in leukemic cells; synergy with the antitumoral activity of many cytostatics; independence of its efficacy on pRb, p53 and Bcl-2 expression; lack of interference with the most frequent multidrug resistance proteins (P-glycoprotein and MRP-190); and a strong antiangiogenic activity.