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Whereas most epithelial tissues turn-over and regenerate after a traumatic lesion, this restorative ability is diminished in the sensory epithelia of the inner ear; it is absent in the cochlea and exists only in a limited capacity in the vestibular epithelium.
Epithelial cadherin (E-cadherin) is expressed by most epithelial tissues.
MICA is transcribed in keratinocytes, endothelial cells, fibroblasts, monocytes, epithelial cell lines and reputedly in most epithelial tissues [ 4].
Most epithelial tissues have some plasticity in their differentiation status and can convert between epithelial and mesenchymal if the right signals are given.
E-cadherin is expressed by most epithelial tissues, and certain proteins expressed in cancer cells are also related to E-cadherin, such as Snail, Smad2, and Smad3.
E-cadherin (CDH1) is expressed on the cell surface in most epithelial tissues [ 1, 2], and evidence is rapidly accumulating that the mammalian E-cadherin gene product plays a role in epithelial tumourigenesis [ 3].
Similar(52)
But in larger immuno-histochemical studies it has become clear that Trask is widely expressed in most normal epithelial tissues, quite abundantly in some, and similarly widely expressed in epithelial cancers without significant evidence of overexpression [10], [11].
Epithelial cell adhesion molecule (Ep-CAM is a typEp-CAM ismembrane glycoprotype of Mr 40 000 Da, expressed In mostransmembranehelial tissues on the basolateral surface.
Most normal epithelial tissues, including breast, prostate, lung, and ovary, express high levels of nuclear p27 protein, especially in the terminally differentiated layers.
The histoblood group Lewis antigens are found in most human epithelial tissues, where they are expressed at the terminal part of glycolipid and glycoprotein glycan chains [ 59].
The simultaneous expression of multiple CEACAM family members by most human epithelial tissues, including GPI-anchored and transmembrane forms in different splice variants, has hampered progress in deciphering the molecular signaling connections initiated by CEACAM-mediated cell-cell interactions.
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