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One source for the differences could have been the fact that most current assays measure a global chemotaxis index that depends on multiple aspects of neutrophil migration, including the fraction of the neutrophil population that are able to move, the alterations of speed, directionality or persistence of migration towards chemoattractant gradients.
These also call for improving the assays' amenability towards automation and miniaturization because most current assays still require the use of expensive and complex instrumentation and complex procedures, limiting their potential in rapid diagnosis.
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In most current BMA assays vertebrate primers amplify DNA from a blood meal, commonly an abundant mitochondrial (mtDNA) locus, which is then sequenced and compared to known sequences in GenBank to identify its source.
The new generation of personal genome testing companies is analytically reliable,4 such that most current "genomic assays have high sensitivity and specificity for measured genetic variants" [ 29].
Most of the current assays for proteasome inhibition are based on cell-free assays [18], [19], which require purification of 26S or 20S proteasomes from different sources.
Most current HPV genotyping assays rely on polymerase chain reaction amplification of a 60- to 450-base-pair region of the HPV L1 gene using consensus primers (i.e., targeting conserved regions of the gene).
Theoretically, TSP can be directly incorporated into the design of assays for most current single-marker SNP genotyping methods to provide enabling advances that simplify assay design and development, increase assay throughput, and improve assay automation and genotyping calling.
Theoretically, TSP can be directly incorporated into the design of assays for most current single-marker SNP genotyping methods.
It should be possible to directly incorporate TSP into the design of assays for most current single-marker SNP genotyping methods.
With the exception of the single cell capillary electrophoresis method, most current single cell kinase activity assays lack the capability of looking at more than one kinase activity at a time.
Perhaps there are small differences in the assay conditions that have a large impact on the results, or perhaps our model of regulation of kinase activity by GTP binding is flawed, but the most likely interpretation is that the current assays need refining.
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