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The ultrastructural features of most cells appeared pathological changes.
More dramatically, in the ApoER2-full-GFP-electroporated cortex, most cells appeared dispersed through the CP, the intermediate and the SVZ with very little, if any, cells positioned just beneath the marginal zone (Fig. 2C).
Higher levels of GskA appeared to be present in the perinuclear cytoplasm and, although present, the concentration of GskA in the nucleus in most cells appeared lower than in the cytoplasm.
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Most likely this is because during nutrient depletion, most cells appear to arrest without buds and with a G1 content of DNA, which suggests they are not in S phase.
There is a production of a small number of heterogeneous population of cells that express characteristics of pancreatic tissues; however, most cells appear to be in the pancreatic endocrine precursor stage.
Whereas most cells appear to express both isoforms, cells of haemopoetic origin and endothelial cells only express talin1, and talin2 is not upregulated when talin1 is depleted from endothelial cells (Kopp et al., 2010).
Although SNc astrocytes and microglial cells were labeled for angiotensin or prorenin receptors, most glial cells appeared less intensely immunoreactive than the dopaminergic neurons.
Although astrocytes and microglia were labeled for angiotensin and prorenin receptors in the normal SNc, most glial cells appeared less immunoreactive than the dopaminergic neurons.
Immunohistochemical staining for UCP1 in BAT confirmed our earlier prediction that thermogenesis at P11 is induced to a high level because most BAT cells appeared densely stained at this time.
In the fluorescence images, most of the cells appeared dead after treatment with cisplatin-functionalized silica particles and free cisplatin, Fig. 8c, d.
Moreover, the cells in the extravascular space, most likely tumour cells, appeared to take up and accumulate ICG within cells, as observed with a confocal laser microscope (Fig. 6b).
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