Sentence examples for mortem studies have from inspiring English sources

Genetic and post mortem studies have shown a large number of mitochondrial abnormalities in the substantia nigra pars compacta (SNc) of the parkinsonian brain.

In addition to the major findings of hypofrontality in patients obtained using brain imaging methods [1], numerous post mortem studies have shown alterations in the expression of genes and proteins involved in major energy metabolism pathways [2] [6], and studies of peripheral tissues have also detected metabolic alterations in first onset and drug naïve patients [7], [8].

13 Post mortem studies have indicated an association between diabetes mellitus and decreased melatonin secretion.

Several post mortem studies have demonstrated significantly greater Aβ burden in patients with EoAD than in those with LoAD [ 20– 22], but PET studies have failed to give consistent results [ 23– 25].

Although apoptosis is postulated to play an important role in the pathophysiology of septic cardiomyopathy [5,6], post-mortem studies have demonstrated relatively little cell death, even in cases of undoubted severe septic cardiomyopathy [7,8].

Post-mortem studies have found reduced levels of BDNF in the hippocampus and prefrontal cortex, in those with and without psychiatric conditions.

Case series and post-mortem studies have shown the diagnosis of smear-negative TB to be highly specific [5].

With regard to the cingulate gyrus, earlier post-mortem studies have observed altered neuronal arrangement [6], smaller pyramidal cells [7] and reductions in the oligodendrocytes within this brain region [8].

The lack of a reference standard for definitive diagnosis of CM is associated with misdiagnosis of CM, particularly in African children where post-mortem studies have shown approximately 20% of "cerebral malaria" cases were due to other causes [1], [35].

Estimates of age-adjusted prevalence vary between 1.9 and 4.4 per 100,000 persons although this may represent an underestimate as post-mortem studies have suggested that as many as 5% of cases of clinically diagnosed Parkinson's disease may in fact have MSA.

Post-mortem studies have identified widespread extramotor as well as motor white matter degeneration (Smith, 1960).