The mortality was corrected using Abbott's formula: M c = M t % – M nc % / 100 – M nc % × 100, where Mc = corrected mortality; Mt = mortality of the test; Mnc = mortality of negative control [ 32].
For qualitative variables (e.g., mortality), the χ-test test was used.
The highest treatment with a measured Ag concentration of 62.6 µg/L, however, resulted in 100% mortality of the test animals at a medium concentration comparable to the NOEC determined with effluents obtained from the STPs.
Furthermore, v-ATPase-A and rpl19 also caused high mortality in the test of WCR feeding assays at relatively low dose [11].
The differences in accumulated mortality among the test groups were checked by using contingency tables.
Hence, the number of records per individual equals the number of days (measured from the time of the first observed mortality in the test) until death or censoring.
Another aspect of our study is that serum MDA levels could be used as a biomarker to predict the clinical outcome of septic patients according to the results of ROC curve analysis to predict 30-day mortality, although the test properties (sensitivity, specificity, positive and negative predicted values, and positive and negative likelihood ratios) for MDA to predict 30-day mortality are moderate.
For the correlation between hyperglycemia and hospital mortality, the χ test was used, with the Mann-Whitney U test used for the correlation between GL (as continuous variable) and mortality.
Mortality of those on ART was similar until 2016 for the two strategies, when mortality for the test-and-treat strategy is expected to reach its peak at 0.5%/year and subsequently start to decline, whereas that of the current strategy would continue to rise considerably for at least another 5 years.
Under laboratory conditions in the petri dish experiment, the heterorhabditid nematodes proved a more effective control of P. saucia larvae and increase in time of exposure led to more mortality of the tested nematode species.
