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We found genotypic variation in both sexes for chill-coma resilience, for the rate at which it declines with age, for longevity, for the initial mortality rate, and for the rate at which mortality increases with age.
Virus-induced prokaryotic mortality increases with water depth from 16%±3% (mean±s.e.m., n = 11) in coastal sediments to 64%±3% in mesopelagic sediments (>160 1,000-m depth, n = 41) to 89%±2% in sediments beneath 1,000-m depth (n = 67 Fig. 3)..
Mortality increases with age and comorbidities, and is higher in patients without previous history of PUD [1, 2, 8].
Previous studies have shown that overall mortality increases with lower values of PDRICG and that a PDRICG threshold >14% is a good predictor of survival [20, 21].
For example, adult mortality increases with age, i.e., mortality at age 71 does not drop below the level at age 70 in a given year.
It is well established that injury mortality increases with remoteness from major cities (Danne 2003; Fatovich and Jacobs 2009; Mitchell and Chong 2010), is the second highest cause of death for Indigenous people (Australian Institute of Health and Welfare (AIHW) 2007) and that alcohol consumption is correlated to injury mortality (Demetriades et al. 2004).
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Mortality increased with the length of the HW.
Latent mortality increased with exposure concentration and duration.
In general, haemolymph osmolality increased with salinity and limpet mortality increased with temperature.
Mortality increased with age (p=0.003) and was inversely related to the CRP peak (p=0.007).
Finally, pollen beetle mortality increased with ground-dwelling generalist predator abundance and soil clay content.
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