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During the maturation and dispersal period, described by the on-off function M t), juvenile mortality increases to j+µ.
At the same time, standard deviation and skewness of the distribution increase dramatically (Fig. 3B,3C), giving eventually rise to a broad and highly asymmetric distribution, and mortality increases to up to 20%.
Most importantly, once breast tumor cells metastasize to bone marrow, mortality increases to 70% in comparison to 40% in the breast cancer patients with no tumor cells in their bone marrow [52], [53].
Mortality increases to almost 70% if diaylsis therapy is initiated.
When controlling for endogeneity, the reduction in in-patient mortality increases to 87%.
Predicted premature mortality increases to a little over 5,000, and based on the concentration-response function used, ranges from 2,400 to over 6,300.
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Mortality increased to 46.2% when norepinephrine dose reached 0.75 µg kg per minute.
In combination, however, the effect was much greater as the RGR was reduced to 0.13±0.03 and the mortality increased to 63±7.1% (Fig. 4c, Table 2).
When these two doses of Mir1-CP and Bt-CryIIA were combined, the RGR value was reduced to 0.22±0.05 (Fig. 1c) and the percentage mortality increased to 50±5.4%, which was somewhat higher than those of the individual treatments (Table 2).
However, when 0.06 ppm Mir1-CP and 0.5 ppm of Bt-CryIIA were combined, the RGR was far lower (0.19±0.03) and the mortality increased to 57±6.3% (Fig. 2c, Table 2).
Mortality increased to 63% in 1 year.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com