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From 1980 2011, annual drug overdose mortality increased from 2.7 to 13.2 deaths per 100,000 population.
Average time scales of carbon recovery increased by 40 years when mortality increased from 30%to65%5%, and another 10 years when mortality rates exceeded 90%.
As the inoculum increased from 103 DNA copies to 104, 105, 106, mortality increased from 20% to 60%, 80 % and 100, respectively.
Sepsis-3 criteria clearly categorized septic patients along a spectrum of severity, since mortality increased from 7% in no-dysfunction-infected patients to 25 and 46% for sepsis and septic shock patients, respectively.
Hospital mortality increased from 13%% for the younger patients (age < 40 years) to 38%% for the older patients (age ≥ 80 years), while Simplified Acute Physiology Score II (without age-related points) increased only from 36 (age < 40 years) to 43 (age ≥ 80).
Mortality increased from 7.1% before to 16.8% after tracing patients [19].
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They are also consistent with other epidemiological analyses, which suggest a continuous gradient of risk of mortality, increasing from lower to higher A1C levels.
As expected, there were age-related trends, with 28 day and one year mortality increasing from 4.1% and 13.1% in those aged 45 49, up to 14.9% and 41.8% in those aged ≥85 years.
US population based in-hospital mortality increased modestly from 1.9 to 2.1 per 100,000 (P<.001), with hepatorenal syndrome as the strongest predictor of death (odds ratio, 9.5; 95% confidence interval, 8.4 10.7).
Maternal mortality increased dramatically from 390 per 100,000 births in 1990 to 790 in 2008 [ 19].
In men, mortality increased rapidly from the 1870 cohort to the 1905 cohort.
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