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Pre-hospital mortality has often shown to be stronger related to SES than case-fatality [ 5– 8].
To avoid this, perinatal mortality has often been used as measure, combining the stillbirth rate with the early neonatal mortality rate.
Although several studies showed inverse associations with CVD, no relationship between diet quality scores and cancer mortality has often been reported.
Infant mortality has often been analyzed according to two components, the birth weight distribution and birth weight-specific mortality; these are believed to reflect maternal health and perinatal health care services, respectively [ 1- 3].
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Sex differences in bladder cancer mortality have often been reported 1– 3, which may be partially explained by the higher proportion of unfavorable pathology in female patients.
Although the mortality reduction is currently believed (even from screening supporters) to be about half of the initially claimed 30-35% [ 20– 22], the contribution of mammography screening to the general mortality decline has often been considered substantial [ 23, 7].
However, based on the results of the aforementioned Australian cohort study [ 2], which identified a lower than expected risk of long-term neurologic sequelae among surviving children and a smaller reduction in mortality than has often been assumed based on screening, some previous cost-effectiveness analyses may have overestimated the clinical benefit of newborn MCADD screening [ 24].
The high cardiovascular mortality in Eastern Europe has often been attributed to poor diet, but individual-level data on nutrition in the region are generally not available.
Previous research about IDU and risk of hospitalizations and mortality [ 8– 10] has often been limited by lack of information about important clinical variables, such as dementia, or by analysis of small and selected samples.
The influence of hyperthermia on trauma mortality has not often been addressed.
1 The effect of VAS on mortality has most often been ascribed to prevention and treatment of vitamin A deficiency. 29 We have previously found poorer vitamin A status in children of non-educated mothers.
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