Not only did climate variables do a poor job of predicting supra-annual mortality cycles, infant population cycling in the two chimpanzee communities were not even in phase.
Rather, infant mortality cycles appeared to self-organize in response to the ontogeny of social play.
This leaves behavioral mechanisms as the only plausible explanations of how predation might drive infant mortality cycles.
There are also good reasons to doubt that predation was a major driver of the observed infant mortality cycles.
And, as mentioned above, there was little support for an association between extreme climate fluctuations (e.g. El Niño) and peaks in mortality rate, as one might expect if starvation were a major force structuring infant mortality cycles.
Note that the mortality cycles estimated refer to the day of admission, so for example, NSW, the ACT, the NT, VIC, SA and WA all have higher mortality associated with weekend admissions.
The cell morphology, cell mortality, cell cycle arrest, and functional status of caspases, upon the treatment by these biomaterials, are characterized.
The five-step maternal mortality surveillance cycle provided the framework for discussion.
The facility-based maternal death review approach is not novel, and neither is the five-step maternal mortality surveillance cycle that underpins it.
In other words, infant mortality rates would cycle on a period of roughly one year.
