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Relationships between amyloid deposition and clinical profile or morphometric changes of islet endocrine cells were examined by correlation analysis.
In addition to studying morphometric changes of the lung parenchyma following high oxygen treatment, changes to the accompanying lung vasculature were also examined.
Where small-intestinal biopsies have been examined in adults, the morphometric changes of EED can be quantified, but they do not appear to correlate well with functional biomarkers [ 2].
We can relate our findings to sepsis-induced myopathy but not to CIM, since the observation period was too short and we did not find the morphometric changes of muscle fibres characteristic of CIM, and also there was no significant change of mean values of densitometric scanning of immune reactive bands of myosin normalized to actin.
A small intestinal biopsy study of adult volunteers in Lusaka, Zambia and Soweto, South Africa found that all the Lusaka residents but none of the Soweto residents had morphometric changes of EED; at the time of the study, sanitation coverage was 59% in Lusaka [ 62] and 99.9% in Soweto [ 63].
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The structural data relate to a morphometric change of the neuronal density in this region, whilst the functional imaging data are related to the neuronal activity in this area.
The finding of morphometric changes in the brains of migraineurs may be of fundamental importance because migraine has so far been considered a condition with abnormal brain function but completely normal brain structure.
The aim of this study was to describe the microscopical lesions and to quantify morphometric changes in the lamellae of horses with insulin-induced (n = 4) and oligofructose (OF -induced laminitis (n = 4) cOF -inducedh normalaminitiss (n = 4).
The morphometric changes showed an increase of the vessel volume fraction and separation and a decrease of vessel connectivity density during the vascular healing process after SCI.
An specific study of morphometric changes in TN [14] revealed brain matter volume reduction in the primary somatosensory and orbitofrontal cortices as well as anterior cingulate, cortex insula, secondary somatosensory cortex, thalamus, putamen, caudate nucleus, dorsolateral prefrontal cortex, precuneus and cerebellum.
The automated spine analysis algorithms presented in this study provide a much-needed tool for the objective evaluation of morphometric changes that occur with synaptic plasticity, normal development and aging, and with neurodegenerative disorders that impair normal cognitive function.
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