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TORC2 may also regulate cell growth, but importantly also appears to regulate cell morphology by mediating actin cytoskeletal modifications in response to growth factors.
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JA can determine the rice spikelet number and morphology through the JA signaling pathway or by mediating stress signals, subsequently affecting the grain yield (Cai et al. 2014; Kim et al. 2009).
LDD is often associated with unusual behavior of the standard vector inferred from plant dispersal morphology, or mediated by nonstandard vectors.
Thus it is likely that the progressive loss of the columnar morphology is mediated by the decreased proliferation, the gradual loss of ciliated cells that were differentiated before the deletion of Sox2 in their progenitors (Clara cells) and the rapid loss of differentiated features of Clara cells after deletion of Sox2.
The profound effect pUL135 exerts on cellular morphology was mediated by two distinct mechanisms acting independently through talin and ABI1/ABI2.
TORC2, which includes Rictor, is implicated in the control of cell morphology mediated by regulation of the actin cytoskeleton [33].
Furthermore, other processes such as cellular localization of proteins and cellular morphology processes are mediated by protein binding events and are pivotal for cells and the functioning of the body.
Such unique growth morphologies are mediated by the asymmetric surface diffusivities on the GaAs (001) surface.
Specific interactions, miscibility, and self-assembly morphologies mediated by hydrogen bonding interactions of this new A-B/C type blend, were investigated in detail.
Lambrot et al. (2009) also recently found an MEHP-induced decrease in the mRNA expression of AMH by human fetal testis in vitro at 10−4 M. Whether the MEHP-induced alteration of Sertoli cell morphology and function is mediated by effects on testosterone, a reduced gonocyte population, or a combination remains to be established.
Moreover, the aberration in intestinal epithelial cell morphology and actin filament organization mediated by TcdA may, at least, partially account for the severe intestinal mucosal disruption found in C. difficile-induced disease.
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