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Nilsson and Pelger's experiments assume that morphological functions can be optimized separately without tradeoffs (Hansen 2003) and assume a particular pathway of morphological evolution.
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Thus, studies have had to rely on the largely unsupported assumption that number of functions can be measured indirectly, by using number of morphological, physiological, and behavioral partsas a proxy.
Co-operation between functions can be limited.
Functions can help.
Functions can map complex motions too.
MRI provides exquisite morphological information, and in addition, ventricular and valvular function can be quantified.
Thus, the diastolic function can be accurate and easily evaluated using morphological and quantitative parameters similar to those obtained by TTE, which are so familiar to general cardiologists.
Two impediments to understanding the role of Hox genes in morphological evolution have been the limited number of organisms in which Hox gene function can be studied and the paucity of known Hox-regulated target genes.
Gene function can diversify in several ways.
Collectively, these findings suggest that abnormalities in macrovascular endothelium-independent function can occur without concomitant abnormalities in macrovascular endothelium-dependent function, and that macrovascular endothelium-independent function is related to morphological changes in the vasculature.
This morphological damage can alter the function of the epithelial barrier, allowing toxin absorption through the large intestine.
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