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Malformations of the cortex are congenital morphological defects stemming from an intrinsically abnormal developmental process.
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The results showed good crystalline behaviors and morphologies without any identifiable morphological defects.
These mice were viable, with no apparent morphological defects.
These observations indicated that the rel2 mutant has multiple morphological defects.
dC1GalT1 mutant larvae have also been shown to have morphological defects in the CNS.
Embryos were scored for morphological defects and Sox9 was visualized via in situ hybridization.
Exposure induced resorption, gross morphological defects, and skeletal abnormalities, without effect on litter size or maternal or offspring weight.
Surprisingly, these mice did not exhibit any apparent morphological defects in the development of normal retinal vasculature.
These adults were examined for external morphological defects.
We discovered morphological defects in eyes, wings and legs.
Rad21+/−mice were viable and developed to apparently normal adulthood without morphological defects.
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