Sentence examples for morphological abnormalities that from inspiring English sources

Initial experience with CT has shown that CT can detect morphological abnormalities that explain abnormally high transprosthetic Doppler velocities [ 5, 7, 8].

Although the pathogenesis of migraine is far from completely understood, clinical neurophysiology and neuroimaging studies in recent decades have disclosed subtle functional and morphological abnormalities that manifest during the interictal phase and distinguish migraineurs from normal healthy subjects [1 3].

This failure is the result of morphological abnormalities that arise during various stages of floral development.

We also performed ultrasonography to exclude the patients who had any morphological abnormalities that might affect the LSM results.

Furthermore, we demonstrate that mitochondrial dysfunction precedes the mitochondrial morphological abnormalities that are seen at the ultrastructural level.

The two small peaks that follow this period represent morphological abnormalities that arose during alevin development (ie; curved backs).

Even in this case, the macroconidia of ΔBAR1 strains displayed morphological abnormalities in that they were significantly shorter and did not display the slender, canoe-shape morphology typical of F. graminearum macroconidia (Fig. 3A; Table 1).

However, F2 hybrids and backcrosses were of poor viability and some of them showed morphological abnormalities, suggesting that there are barriers to introgression of genetic material between the species.

Dgp71WD homozygous or hemizygous adult flies, however, did not show the same reduced lifespan or any morphological abnormalities, suggesting that these phenotypes are not the result of a lesion in the Dgp71WD gene.

In contrast to Lhx6 null mutants (Liodis et al. 2007), compound heterozygous Lhx6 LacZ/− and homozygous Lhx6 LacZ/LacZ animals survived to adulthood, were fertile and showed no gross morphological abnormalities, indicating that Lhx6 LacZ rescues the post-natal lethality of Lhx6-deficient mice.

While our homozygous knockout mice show typical embryonic malformations previously described for the lack of the Nkx2-5 gene, hearts of heterozygous adult mice show moderate morphological and functional abnormalities that are sufficient to sustain blood supply demands under homeostatic conditions.