Sentence examples for morphological abnormalities at from inspiring English sources

The cdc42-1 strain contains a temperature-sensitive allele of CDC42, resulting in minor morphological abnormalities at the 23 24°C and growth arrest as unbudded cells at 37°C [8].

The phenotype obtained in mbnl2 morphants was demonstrated to be specific and dose dependent, and included morphological abnormalities at the eye and skeletal muscle levels.

The Sgce-null mice did not show any morphological abnormalities at any of the ages analyzed, while the Sgca-null mice and Sgca-; Sgce-null mice showed extensive pathological alterations already at 2 months of age.

Around 15% of EP mutants are expected to show peri- or postnatal, rather than earlier embryonic, lethality, and such embryos are likely to exhibit no obvious morphological abnormalities at E14.5 E15.5.

Interestingly, trout eggs obtained by hormonal induction, which result in alevins with a high percentage of morphological abnormalities at the yolk-sac resorption stage, also show a dramatic increase of apoc1 [ 18].

We have generated a zebrafish knockdown model in which we demonstrate that a lack of mbnl2 function causes morphological abnormalities at the eye, heart, brain and muscle levels, supporting an essential role for mbnl2 during embryonic development.

Additionally, these strains may model the dosage-dependent nature of SMA [1], [2] as the developmental arrest associated with these animals correlates with the extent of morphological abnormalities observed at the NMJ.

However, ERGs in the Adam9−/− mice appear less severely disturbed than those of β 5 −/− mice, whereas β 5 −/− mice lack the morphological abnormalities observed at the RPE-POS junction in Adam9 −/− mice.

The percentage of normal (i.e. without morphological abnormalities) alevins monitored at yolk-sac resorption (YSR) was used to characterize the egg quality of each individual female.

In this study, we have described such an approach and present the results of a pilot study examining the morphological abnormalities in embryos at E14.5 from 34 recessive lethal knockout lines of the IKMC-coordinated gene-knockout screen.

Furthermore, we demonstrate that mitochondrial dysfunction precedes the mitochondrial morphological abnormalities that are seen at the ultrastructural level.