Sentence examples for morphogenetic cues from inspiring English sources

The protein encoded by this gene, polycystin-1, is a renal epithelial cell membrane mechanoreceptor, sensing morphogenetic cues in the extracellular environment, which regulate the tissue architecture and differentiation.

Polymeric delivery systems may, instead, mimic certain sequestration and binding characteristics of the extracellular matrix and lead to the provision of therapeutic molecules at therapeutically efficient local concentrations [V], in the form of spatial gradients (d[V]/dx) and temporal gradients (d[V]/dt), and in combination with other morphogenetic cues.

In vivo, embryogenesis is a highly orchestrated process involving the interaction of several signaling networks that produce local morphogenetic cues that determine cell fate and tissue organization[1].

This would allow for efficient and rapid transmission of morphogenetic cues, simply by exploiting the rate-limiting role of auxin signaling for plant development.

Moreover, endogenous bioelectric signals (patterns of resting potential in all tissues) encode a set of morphogenetic cues that provide patterning control during regeneration (Levin, 2007; Levin, 2009), development (Adams, 2008; Levin, 2012a) and wound healing (Nuccitelli, 2003; McCaig et al., 2005; McCaig et al., 2009).

Studies of coral settlement and metamorphosis have indicated that the inductive morphogenetic cue is exogenous/environmental and, whilst the exact structure of the metamorphosis inducing morphogen remains elusive, lipopolysaccharides are prime candidates [ 13] suggesting that cell surface recognition by coral larvae may be mediated by lectins.

Proper development and function of the heart is dependent upon an intricate network of molecular cues and morphogenetic events that are highly conserved among vertebrates (Harvey, 2002; Srivastava and Olson, 2000).

Hypoxia acts as a positive external positional cue controlling morphogenetic and biochemical adaptations to protect the stem cell niche.

Besides photoprotection, UV radiation also triggers safeguards to anticipate other stressors such as heat or drought, and mediates in developmental cues such as morphogenetic responses to shade/light or interactions with other organisms [ 18- 22].

During the dormant phase, crosstalk between the dermal papilla and the hair follicle stem cells contributes to the threshold of activating cues (Wnts, bone morphogenetic protein (BMP) inhibitors and TGF-βs) necessary to shift the stem cells from a quiescent to an activated state [ 36- 45].

First, etsrp+ populations in the head, trunk and tail follow distinct developmental and morphogenetic programs in response to local cues.