Sentence examples for morphogenesis and synaptic from inspiring English sources

Neuronal time-lapse imaging, behavioral analyses, and electrophysiological recordings from genetically modified mice were used to show that WAVE-1 signaling complexes control aspects of neuronal morphogenesis and synaptic plasticity.

Highly enriched in the dendritic spine, neurabin is important for spine morphogenesis and synaptic formation.

Prostaglandins induce glial release of glutamate [18]; glutamate can then act on neurons to regulate spine morphogenesis and synaptic plasticity.

Two non-receptor tyrosine kinases involved in neurite outgrowth, dendritic spine morphogenesis and synaptic plasticity, the focal adhesion kinase (FAK) and C-Src, also co-immunoprecipitated with Dp71.

Because of its spatiotemporal expression pattern, CPD1 has been proposed to play an important role in brain morphogenesis and synaptic development.

Dp71-DAPC is also associated with key signaling proteins such as FAK, c-Src and Grb2, proteins known to contribute to neurite outgrowth, dendritic spine morphogenesis and synaptic plasticity [24] [26].

Here, Cagla Eroglu and colleagues report that neuroligins a family of cell adhesion molecules expressed by astrocytes can bind to neurexins expressed on neurons to regulate astrocyte morphogenesis and modulate synaptic density and function.

For example, one of the most conserved Drosophila clusters includes lush (table 1 and fig. 5), which encodes an OBP involved in social aggregation and mating behavior (Xu et al. 2005), but also Shal (a potassium channel), ash1 (involved in the ovoposition and oogenesis), asf1 (dendrite morphogenesis), and tey (synaptic target recognition).

In conclusion, the Neph orthologs in D. melanogaster and C. elegans constitute signaling modules that mediate cell cell recognition and cell sorting in tissue morphogenesis, axonal pathfinding, and synaptic plasticity (Fischbach et al. 2009; Huber and Benzing 2005).

Upon its activation in neurons, Akt phosphorylates different substrates, which in turn regulate diverse processes of neuronal development, including morphogenesis, dendritic development, synapse formation and synaptic plasticity (Yoshimura et al., 2006), all of which are altered in FASD.

Dysregulation of the Wg nuclear import pathway (FNI) provides a plausible mechanism to explain synapse development defects underlying the FXS disease state, with established roles in activity-dependent modulation of synaptic morphogenesis and neurotransmission (Korkut and Budnik, 2009).