Sentence examples for more rostral brain from inspiring English sources

Given that local within-block probabilities remained constant across conditions, any change in MMN size was considered a reflection of more rostral brain regions enabling a longer time scale (across-block) representation of event-probability extraction.

It is notable in this regard, that even prolonged and strong stimulation trains applied to more rostral brain sites such as the posterior hypothalamus (that forms part of the ascending activating system but is not thought to be involved in pacing state alternations during sleep per se) caused no such temporary depression of the ongoing alternations.

The exogenous protein progressively spread toward more rostral brain regions where it could be detected within axonal projections.

This possibility is at least suggested by data available for L-type BSE, which shows a preferential distribution of abnormal PrP in more rostral brain regions (4, 12 ).

Enhanced expression of hα-syn in the rat MO has been shown to trigger its interneuronal propagation toward more rostral brain regions [ 12].

α-Syn spreading from the caudal brainstem toward more rostral brain regions was triggered by protein overexpression in the rat MO and compared under experimental conditions characterized by absence of neurodegeneration vs. a loss of α-syn-containing neurons.

Further analysis of the spatial and temporal progression revealed that with increasing time p.i., the deposition became more prominent in rostral brain regions, although caudal regions continued to be affected.

Greater qualitative discrimination was possible examining more rostral areas of the brain, and particularly the cerebellum, despite this area not necessarily displaying the greatest amount of PrP [ 43].

This regional difference in responsiveness to inflammatory stimuli is also evident in EAE, which targets the spinal cord rather than more rostral regions of the brain, such as the forebrain (Sun et al., 2004).

Various researchers have also reported that DA exposure causes prolonged neuroexcitation and extensive degeneration in the hippocampus as well as more rostral areas of the brain, including the septum and olfactory bulb (Peng and Ramsdell 1996; Peng et al. 1994; Scallet et al. 1993, 2004).

At 9 months post injection aggregates with phosphorylated alpha-synuclein were more abundant, equally present in both brain hemispheres, and had spread to more rostral and caudal areas in the brain (Figs.  4 and 5, and Additional file 3).