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Thus, more genes (maybe encoding other Nups) are involved in this hybrid incompatibility.
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More genes may be found.
However, these genes maybe partly or completely epigenetically inactivated.
This might imply that TCR is more active in CGI-genes, maybe due to their higher expression in germ cells.
The God gene maybe?
More important, maybe.
More transparent, maybe.
Yay, more books (maybe)!
Maybe more, maybe less.
Except Gene Roddenberry, maybe.
The appearance of a new splicing site could lead to a qualitatively different gene product or, maybe more relevant in our model, to the overexpression of normal transcripts [31].
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