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Looking at these same genes more exhaustively, and considering the more than 20,000 genes in the human genome, there should be many more genes identified with MAP which might contribute to the disease susceptibility in a multifactorial and tissue-specific way.
Not only were more genes identified, the expression of which was induced upon infection, also the fold changes were found to be increased after vRNA clearance.
In addition, there were more genes identified in the ino F comparison than in the ino E comparison, which indicates that there are several identified genes expressed in later stages of ovule development.
Taken together, consistent findings of more genes identified by paired t-tests in welders than in controls suggested there were alterations of global gene expression profiling in the whole-blood total RNA in response to acute metal-fume exposure.
The number of SNPs between M06-24/O M06-24/Oreference genomes wandmuch lower then those freferencether three genomes (Table 4), even though there wase slightly much genes identified in M06-24/O.
Also, at lower extent, chromosomes 2, 6 and 12 had more genes identified in the RGP than in the Met library, whereas genes from chromosomes 11 and 13 showed an inverted pattern.
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One of the more interesting genes identified encodes the OmpC porin protein (PBPRB1639).
There are now more than 35 genes identified in which variable number tandem repeats cause disease [ Pearson et al., 2005].
The study of more than 22,000 genes identified a gene expression signature related to invasion that is differentially regulated between FLSs from DA and DA.F344(Cia5d) rats.
The gene list employed in this study was selected from the list of more highly expressed genes identified in somatic embryos in the present study.
Perhaps more importantly, many genes identified in this study have been previously suggested to be expressed specifically or preferentially in the brain.
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