Thus, the brain provides a more extensive matrix for damage than is seen in many other organs.
Podosomes, for example, are typically small (0.5 1 μm in diameter), numerous (20 200 per cell) with a short half-life and producing only shallow matrix degradation while invadopodia are much larger structures (8 μm in diameter) that are less abundant in cells (less than 10 per cell) have a much longer half-life and are capable of much deeper and more extensive matrix degradation (Linder, 2009).
The MLCs exhibited a much more extensive extracellular matrix than monolayer cell cultures.
The onset of dissolution for SIMFUEL and the doped specimens was initiated by tetragonal distortions of the cubic matrix and was accompanied by a deeper more extensive oxidation of the UO2 matrix.
Therefore, our results obtained from the matrix domain of the binomial matrix (B^{r,s}=(b_{nk}^{r,s})) are more general and more extensive than the results on the matrix domain of the Euler matrix of order r.
Moreover, G m ( 1, − 1 ) = Δ m, G 2 ( 1, − 1 ) = Δ, G 3 ( 1, − 1 ) = Δ 2. So, our results obtained from the matrix domain of the m th order generalized difference matrix G m are more general and more extensive than the results on the matrix domain of B ( b 1, b 2 ), B ( b 1, b 2, b 3 ), B ( b 1, b 2, b 3, b 4 ), B ( b 1, b 2, b 3, b 4, b 5 ), … , Δ m, Δ 2 and Δ.
Regardless of the initiating stimulus, paroxysmal AF promotes further AF and stretch, leading to more extensive fibrosis and increased extracellular matrix deposition that ultimately slows or blocks intra-atrial conduction as a part of the adaptation to high atrial rates.
So, our results obtained from the matrix domain of the binomial matrix (B^{r,s}=(b_{nk}^{r,s})) are more general and more extensive than the results on the matrix domain of the Euler means of order r.
Hepatocytes on compliant or lower adhesive matrices exhibit more extensive intercellular interactions, histotypic cytoskeletal organization and physiological phenotype (Mooney et al., 1992; Powers et al., 1997).
In the crack propagation process, tensile cracks of rock matrix (TCRM) are far more extensive than other crack types, and shear cracks of bedding planes (SCBP) determine the crack orientation.
In particular, there was a differential activation of the MAP kinase pathway and, in VAT, a more extensive remodeling of cell structure and extracellular matrix and an important participation of its stromal environment.
