Sentence examples for more effector cells from inspiring English sources

Similarly, more effector cells lead to more activated APCs and thus to more effector cells.

The initial innate response would be expected to include activation of macrophages, neutrophils, and Langerhans cells as well as pro-inflammatory signaling to recruit more effector cells (leukocytes).

It is not clear whether these effects were due to the generation of more effector cells from naive T cell populations, the expansion of memory T cell populations, or both.

In addition, Inf patients tended to have more effector memory cells (TEM) and fewer effector cells (TEFF) than did ENs giving significantly smaller TEFF ∶ TEM ratios.

Surprisingly, data from this study found that not only were there more effector memory cells in Inf and CLInf patients, these cells had a higher mean expression of IL-7Rα (CD127), a finding that differs from what has been seen in chronic viral infections in humans [25], [38], [40] and in mice [70], [71].

Further study at later time points, when more effector Th17 cells would be present in the joint, would be required to determine whether the reported synergistic induction of ADAMTS-5 from macrophages by TL1A and IL-17 occurs in AIA (56).

CD8 T cells supplied with CD4 help at the time of initial priming generate more efficient effector cells upon secondary challenge [20], [21].

Recent work by Segal and colleagues in C57BL/6 mice also suggested that neutrophils may be more important effector cells in the brain compared to the spinal cord.

TEM cells are first responders that rapidly produce high levels of effector cytokines, while TCM cells have a high proliferative capacity and, thus, produce more secondary effector cells that reinforce the host response to foreign insults.

The isolated NK cells were much more potent effector cells than PBMC, and all samples showed 65.3 ± 2.6% and 79.4 ± 1.6% cytotoxicity at E/T ratio of 8 and 32, respectively.

Compartmentalisation of ESAT-6-specific IFN-γ-secreting cells at the pericardial disease site exclusively in HIV-1-uninfected patients reflects the presence of more differentiated effector cells, congruent with our flow cytometric findings, and may indicate the contribution of these cells to host defense in containing MTB at sites of replication 11.