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Exact(5)
We further observed that migration was directional: 2 3 times more cells migrated toward the layer above, i.e. (towards a higher concentration of oxygen and nutrients) than toward the layer below (Fig. 4G).
Differences in cell migration rate over these two types of collagen substrates were observed at both ages, and all in all, three times more cells migrated over type I/III collagen than over type IV collagen.
When confluent monolayers of cells were untreated with the extracts, MCF-7 cells could migrate to the gap after 24 h and more cells migrated after 48 h.
As culture progressed, more and more cells migrated into the pores, attached to each other, and/or to the pore edge and formed a circular membrane along the edge.
Significantly more cells migrated into the upper chamber in a dose response dependent manner in the presence of HMC-1 exosomes, with the highest doses being statistically significant compere to the control and with a correlation coefficient of 0.91 (p <0.0001).
Similar(55)
NGF recruited more cells migrating through the membrane than culture medium alone (Fig. 3b).
The result is a fatty plaque, which can continue to grow as more cells migrate into it.
Similarly, the transwell migration assays showed that more CNE2 cells migrated through the membrane compared with the cells that had lower expression of SOX4 after 16 h and 5(d)).
During wound healing assay, more A549 cells migrated to the scratch in the cell monolayer when these cells were transfected with pre-miR-19a/b, while A549 cell mobility was significantly inhibited by anti-miR-19a/b transfection (Fig. 4F and 4G).
Strikingly, 4-times more Th1 cells migrated towards CCL4 after stimulation with anti-CD3/anti-CD28/anti-CD152 companti-CD3/anti-CD28-stimulatedsTimulated T cells.
CST3-His expression in HEK293 cells resulted in adhesive growth (Supplementary Figures S1B and C), indicating that the more adhesive HEK293-CST3-His HEK293-CST3-His HEK293-CST3-Histheir growth was retarded more than the cellsHis cells (SupplemigratedFigure S1D).
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